THE REAL VALUE OF NEVIRAPINE

Over the past six months, a simple drug has become the centrepiece of a major political debacle in South Africa with all the intrigues and more twists than a Jeffrey Archer novel. Health professionals, AIDS activist, scientists, religious leaders, trade unionists and senior politicians have all entered into the fray arguing the merits of the drug, its benefits, and toxicities. It has been condemned as "the drug from hell' by AIDS dissidents while heralded as a "godsend" by many health professionals. Amidst all the rhetoric, there have been few attempts to decipher the real value of the drug except through public slanging matches played out in the courts and in the media.

How does nevirapine work?

The human immunodeficiency virus (HIV) uses the enzyme reverse transcriptase to incorporate its own genetic material into host cells, which then allows it reproduce freely. Nevirapine, one of a class of drugs known as non-nucleoside reverse transcriptase inhibitors, prevents this enzyme from functioning. This results in a reduced amount of virus in the body and an increase in the CD4 cell (T cell) count, improving the host's immune function, and thereby reducing the risk of new and opportunistic infections, and death.

When is nevirapine used?

Nevirapine has been widely used in adults as one of a combination of drugs to treat established HIV infection. It has a special role in the prevention of mother to child transmission (MTCT) of HIV, as it is effective when given alone as a single dose to the mother at the beginning of labour and one dose administered to the baby within 72 hours of birth. Nevirapine given to HIV-positive pregnant women rapidly crosses the placenta into the fetus with its effects lasting through the first week of life. The timing of its delivery to the mother during labour is important as up to two-thirds of infants born with HIV are infected in the birth canal.

Is nevirapine effective?

Clinical trials have confirmed the efficacy of nevirapine in preventing mother-to-child transmission (MTCT) of HIV. No trials are available comparing nevirapine with placebo. The Ugandan HIVNET 012 study involving 626 women showed that nevirapine was able to reduce MTCT by 47%, with only 8.1% of infants exposed to nevirapine acquiring HIV at birth.1 Almost all the babies were breast fed, resulting in ongoing exposure to HIV. Despite this, the benefits of nevirapine were maintained with a 42% reduction in transmission at the age of 12 months. 2

In the South African Intrapartum Nevirapine Trial (SAINT), 1306 mother/infant pairs were randomised to either nevirapine during labour and post-delivery, or multiple doses of AZT/3TC during labour and for one week after delivery to mother and baby. In both treatment arms, about 40% of infants were breast-fed. Eight weeks after birth, there was no significant difference observed between the rate of HIV infection or death across the two treatment arms, with a rate of 14.3% in the simpler nevirapine arm and 12.5% in the more involved and expensive dual therapy arm.3 For pregnant women already receiving highly active antiretroviral therapy, there appears to be no additional advantage of adding nevirapine to an existing regimen.4

What about drug resistance?

Drug resistant mutations of HIV are usually naturally occurring variants that are present at low frequency before the use of nevirapine and which are then selected and expanded when the drug is introduced. However, resistance does not impact on the efficacy of the treatment to prevent MTCT, and resistant mutations wane over 12-24 months. Resistance to antimicrobials is not unique to HIV and a recent study,5 reaffirmed the WHO's view that concerns over drug resistance should not delay the widespread use of nevirapine for preventing MTCT.

Is the drug toxic?

Opponents of nevirapine cite toxicity as an important limitation for its use in MTCT prevention. Long-term use of nevirapine in adults has resulted in significant side effects including life-threatening liver toxicity and skin reactions. Similar adverse events have been reported in health care workers taking nevirapine in combination with other antiretroviral drugs for post-exposure prophylaxis after occupational exposure to HIV. However, in the SAINT study where single doses were given to mother and infant, none of these adverse events were noted, with the commonest, but infrequent, side effect being a mild, self-limiting skin rash.

Is nevirapine affordable and cost effective?

The low cost of nevirapine (< R10) and simplicity of administration offers great advantage over more expensive MCTC prevention treatments, particularly for low-income countries where women generally attend antenatal clinics irregularly or late during pregnancy, and where many deliveries occur in home settings. Further, the manufacturer has offered the drug to some governments at no cost.

The cost effectiveness of nevirapine and of MTCT strategies has been well described in the South African setting, with all studies on the subject consistently showing overwhelming cost benefit for prevention measures. Nevirapine is a minor component of the costs of a total MCTC package. Annually, it would cost the South African government (if it decided not to accept the free offer) about R 6 million to provide the
drug to all eligible mothers and infants. A total MTCT package would cost about R 150 million annually (less than 1% of the health budget), with testing (R15 million), counselling (R60 million) and replacement
infant feeding (R70 million) costs being the major contributors.6 It is estimated that by providing the nevirapine package, the health ministry would save about R360 million annually, by preventing about 31 000 children getting HIV and thus saving on the medical cost of treating the disease (excluding the provision of long-term antiretroviral treatment).7

Current research confirms the benefits of nevirapine for preventing MTCT of HIV. Nevirapine is cost-effective, safe, and has no significant side effects when used for short periods. Drug resistance is a transient phenomenon. The benefits of nevirapine appear to be maintained even in the presence of continued breast-feeding. The challenge now is to translate these research findings into policy and practice in South African and other resource-poor settings.

Dr Haroon Saloojee, Principal Paediatrician,
Chris Hani Baragwanath Hospital, Johannesburg
tel: (011) 4815201; email: 092sal@chiron.wits.ac.za

The full version of this article can be accessed at Science in Africa:
Nevirapine - godsend or drug from hell? http://www.scienceinafrica.co.za/


References:

  1. Guay LA, Musoke P, Fleming T, Bagenda D, Allen M, Nakabiito C et al. Intrapartum and neonatal single-dose nevirapine compared with zidovudine for prevention of mother-to-child transmission of HIV-1 in
    Kampala, Uganda: HIVNET 012 randomised trial. Lancet 1999;354:795-802.
  2. Owor M. The one-year safety and efficacy data of the HIVNET 012 trial. 2001. XIII International AIDS conference, Durban, South Africa, 9-14 July 2000.
  3. Moodley, D. The SAINT Trial:Nevirapine (NVP) versus
    zidovudine(ZDV) + lamivudine(3TC) in prevention of peripartum transmission. 2001. XIII International AIDS conference, Durban, South Africa, 9-14 July 2000.
  4. Brocklehurst P,.Volmink J. Antiretrovirals for reducing the risk of mother-to-child transmission of HIV infection (Cochrane Review). Cochrane.Database.Syst.Rev. 2002;CD003510.
  5. Stringer JS, Sinkala M, Rouse DJ, Goldenberg RL, Vermund SH. Effect of nevirapine toxicity on choice of perinatal HIV prevention strategies. Am.J.Public Health 2002;92:365-6.
  6. Geffen, N. Cost and Cost-Effectiveness of Mother-to-Child Transmission Prevention of HIV. 2000. Briefing Paper for the Treatment Action Campaign.
  7. Skordis, J and Nattrass, N. What is Affordable: The Political Economy of Policy on the Transmission of HIV/AIDS from Mother to Child in South Africa. 2001. Paper presented to the AIDS in Context Conference, University of the Witwatersrand, April 2001.

29-05-2002